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acute ischaemic stroke

in review 6 min read Updated 2026-08-24
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acute ischaemic stroke

Time-critical emergency. Thrombolysis (TNK 0.25 mg/kg or alteplase 0.9 mg/kg) within 4.5 h of onset → EVT for large vessel occlusion up to 24 h with imaging selection. Pre-lysis BP <185/110, post-lysis/post-EVT BP <180/105 for 24 h. No antiplatelets for 24 h post-TNK, repeat CT before starting ASA.

acute ischaemic stroke admission bundle
  • NIHSS on arrival → repeat q1h × 6 h, then q2h × 18 h
  • STAT non-contrast CT + CTA arch-to-vertex (add CT perfusion if >6 h or wake-up stroke)
  • blood work: glucose, CBC, INR/aPTT, electrolytes, creatinine, troponin, lipid panel, HbA1c — do not delay imaging for labs
  • fingerstick glucose → treat hypoglycaemia immediately; insulin sliding scale targeting 7.8–10 mmol/L
  • supplemental O₂ to maintain SpO₂ >94%
  • if thrombolysis-eligible (≤4.5 h): BP <185/110 → TNK 0.25 mg/kg IV bolus (or alteplase 0.9 mg/kg) → BP <180/105 × 24 h
  • if LVO on CTA → thrombectomy pathway (in-house or transfer) → EVT up to 24 h with perfusion selection → BP ≤180/105 × 24 h
  • if no thrombolysis or EVT candidate: treat BP only if >220/120 → reduce ~15% over 24 h
  • if thrombolysed: no antiplatelets × 24 h → CT head to exclude haemorrhage → then ASA 160 mg load
  • if not thrombolysed: ASA 160 mg loading dose immediately
  • minor stroke (NIHSS ≤3) or high-risk TIA, non-cardioembolic → DAPT: ASA + clopidogrel × 21 days
  • statin (atorvastatin 40–80 mg) within 24 h → target LDL <1.8 mmol/L
  • 12-lead ECG
  • telemetry × 48 h minimum → if cryptogenic, arrange prolonged cardiac monitoring (≥14 days) → see Cryptogenic and Young Stroke
  • TTE if cardioembolic source suspected
  • if AF detected → plan DOAC with timing based on infarct size; do not anticoagulate ESUS empirically
  • NPO until dysphagia screen (validated tool, within 24 h)
  • oral hygiene protocol to reduce aspiration pneumonia risk
  • temperature monitoring → treat fever aggressively
  • DVT prophylaxis: pneumatic compression devices day 1 → pharmacological when safe
  • mobilisation assessment within 24–48 h (avoid very early high-dose mobilisation per AVERT)
  • multidisciplinary rehabilitation referral (PT/OT/SLP) within 24–48 h
  • neurological deterioration → STAT CT → malignant MCA oedema or cerebellar mass effect → neurosurgery for decompression or EVD

thrombolysis

alteplasetenecteplase
dose0.9 mg/kg (max 90 mg): 10% bolus over 1 min, 90% infusion over 60 min0.25 mg/kg (max 25 mg): single IV bolus over 5 s
window≤4.5 h from onset/last known well≤4.5 h from onset/last known well
advantageestablished evidence basesimpler dosing, single bolus, non-inferior (AcT (2022))

BP must be <185/110 before lysis. Maintain <180/105 for 24 h after.

post-thrombolysis haemorrhage

Any decline in consciousness, new headache, or neurological worsening → stop infusion (if running) → STAT CT head → draw CBC, INR, fibrinogen → consider cryoprecipitate or fibrinogen concentrate + tranexamic acid. Avoid PCC, platelets, and factor VIIa.


endovascular thrombectomy

Eligible if all four criteria met:

  1. Patient — age ≥18, baseline functional independence
  2. Symptoms — disabling deficit (NIHSS >5), last known well ≤24 h
  3. Occlusion — intracranial ICA, M1 MCA, or basilar on CTA
  4. Parenchyma — ASPECTS ≥6 (small core) or 3–5 (moderate core, selected patients)

≤6 h: CTA sufficient for selection. >6–24 h: requires CT perfusion or multiphase CTA to demonstrate salvageable tissue (DAWN (2018), DEFUSE 3 (2018)).

Procedural sedation preferred over GA unless airway compromise or severe agitation.

EVT does not preclude thrombolysis — give TNK/alteplase first if within window, proceed to angiography.


first 24 hours post-TNK

timeaction
0–1 hneuro checks q15 min, BP q15 min, keep <180/105
1–6 hneuro checks q1h, BP q1h
6–24 hneuro checks q2h, BP q2h
24 hrepeat CT head → if no haemorrhage → start ASA 160 mg
24 h onwardtransition to oral antihypertensives if needed, start secondary prevention
no antiplatelets or anticoagulants for 24 h post-thrombolysis

Antiplatelet therapy delayed until 24-h CT excludes haemorrhagic transformation. This applies to both ASA and heparin (including DVT prophylaxis doses).


blood pressure targets

scenariotarget
pre-lysis<185/110
post-lysis (24 h)<180/105
no thrombolysis, moderate HTN (<220 systolic)do not treat routinely
no thrombolysis, extreme HTN (>220/120)reduce ~15% over 24 h (not >25%)
post-EVT (during and 24 h after)≤180/105; emerging evidence suggests avoiding SBP <140

acute antithrombotic therapy

No thrombolysis given:

  • ASA ≥160 mg loading dose immediately → 81 mg daily
  • Alternative: clopidogrel 300–600 mg load → 75 mg daily

Post-thrombolysis:

  • No antiplatelets × 24 h → CT to exclude bleed → then ASA 81 mg daily

DAPT for minor stroke / high-risk TIA (NIHSS 0–3, non-cardioembolic):

  • ASA 162 mg + clopidogrel 300–600 mg load → ASA 81 mg + clopidogrel 75 mg daily × 21 days (CHANCE (2013), POINT (2018))
  • Alternative (AHA/ESO, not CSBPR): ASA + ticagrelor 180 mg load → 90 mg BD × 30 days
  • Do not extend DAPT beyond 21–30 days → bleeding risk outweighs benefit

Timing by stroke severity — start DOACs over Warfarin:

stroke severitystart anticoagulation
TIA, no infarct on imagingwithin 24 h
minor stroke / small infarctday 3
moderate stroke / moderate infarctday 6–7
severe stroke / large infarctday 12–14

Bridge with ASA until anticoagulation started.


traps

  • Clock starts at last known well, not symptom discovery — a wake-up stroke is not automatically excluded (use perfusion imaging)
  • TNK and alteplase have the same time window but TNK is a single bolus → logistically simpler, especially for drip-and-ship transfers
  • Do not chase mild hypertension pre-lysis — aggressive lowering may worsen ischaemia. Only treat if >185/110
  • ASPECTS is scored on non-contrast CT; ≥6 supports EVT, but 3–5 may still benefit in selected patients with favourable perfusion
  • Orolingual angioedema post-alteplase (1–5%) → stop infusion, H1 + H2 blockers, consider epinephrine
  • Very early mobilisation (<24 h, high intensity) may worsen outcomes (AVERT) — early but gentle mobilisation is reasonable
  • Do not delay lysis for blood work unless on anticoagulants or clinical concern for coagulopathy — a normal-appearing patient with known medication list can be lysed before results return

Key references

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